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Table 1 Sample phenotypic distributions

From: Impact of estrogen receptor gene polymorphisms and mRNA levels on obesity and lipolysis – a cohort study

Name Phenotype

Sample 1 Obesity

Sample 2 Obesity replication

Sample 3a Fat cells

Obese

Women (N = 581)

Men (N = 124)

Women (N = 689)

Men (N = 148)

Women (N = 216)

Age (years)

41 ± 12

45 ± 13

43 ± 11

45 ± 12

38 ± 10

BMI (kg/m2)

43 ± 5

45 ± 5

37 ± 5

45 ± 5

38 ± 5

HOMAIR

5.3 ± 5.7

10.5 ± 16.5

4.5 ± 5.7

8.2 ± 5.5

3.8 ± 2.8

Pl-cholesterol (mmol/l)

5.2 ± 1.

5.2 ± 1.2

5.3 ± 1.1

5.2 ± 1.1

5.2 ± 1.1

Pl-HDL cholesterol (mmol/l)

1.2 ± 0.3

1.0 ± 0.2

1.3 ± 0.3

1.1 ± 0.3

1.2 ± 0.3

Pl-triglycerides (mmol/l)

1.7 ± 0.9

2.3 ± 2.3

1.7 ± 1.0

2.0 ± 1.2

1.6 ± 1.0

Lean

Women (N = 338)

Men (N = 64)

Women (N = 525)

Men (N = 88)

Women (N = 12)

Age (years)

50 ± 4

57 ± 8

39 ± 6

36 ± 11

33 ± 8

BMI (kg/m2)

22 ± 2

23 ± 2

22 ± 3

23 ± 1

22 ± 2

HOMAIR

1.3 ± 0.7

1.6 ± 0.8

1.3 ± 1.0

1.4 ± 0.7

1.4 ± 0.9

Pl-cholesterol (mmol/l)

5.4 ± 1.0

5.7 ± 1.1

4.7 ± 0.8

4.9 ± 1.1

4.8 ± 0.7

Pl-HDL cholesterol (mmol/l)

1.7 ± 0.4

1.4 ± 0.4

1.6 ± 0.4

1.3 ± 0.5

1.6 ± 0.4

Pl-triglycerides (mmol/l)

0.9 ± 0.5

1.5 ± 1.0

0.9 ± 0.5

1.3 ± 1.4

0.8 ± 0.2

  1. Values are mean ± SD
  2. a) 99 subjects overlap between sample 1 and 3.